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Plasma tau, which indicates injury to neuronal axons, is higher among children with cerebral malaria and severe malarial anemia. Patients who survive CM tend to have persistent neurocognitive impairment in survivors of pediatric cerebral malaria and severe malarial anemia. The objective of the current study was to determine whether plasma tau can be a proxy marker of NCI after CM and SMA to limit the use of lumbar punctures to obtain CSF biomarkers.

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